The Human Heart proves evolution wrong!

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1) Neurological communication (nervous system)
2) Biophysical communication (pulse wave)
3) Biochemical communication (hormones)
4) Energetic communication (electromagnetic fields)

This would explain why our heart hurts when we lose some one near and dear to us. And it is now believed that one of the functions of the heart deals with our emotions and attitude. But because heart transplants have become more of the norm for a failing heart. Another unexpected side effect has come up. The people who received the donor heart can often experience:

1) The other person’s memories.
2) The other person’s food cravings.
3) Some of the other person’s attitudes.
4) And some of the fears, hates, and loves of different things.

Picture of brains from under a microscope from inside the heart

This would explain why Christ resides in a person’s heart when they are saved. It also explains why God said that He hardened Pharaoh’s heart towards Moses instead of changing his mind. God knew, even before medical science, that emotion of love and hate reside in the heart and not in our minds. If you would like to do more study on this new area of research. Google these words: Heart brain, Heart brain connection, Heart math , Heart transplant memories. Those who would like to do a Biblical research, just use a Bible search engine and search for the word heart. You will see the word of God start to make sense in this area, when before this information came out, it did not.

Does medical science prove this as well?

When you take an antidepressant. What is the main side effect that is a warning if you try and get off of it all at once? Heart attack. Now are we treating the heart or the brain for depression? If the side effect is a heart attack, and not a brain meltdown, what do you think is most affected by the antidepressant drug? The heart is. So even though I have read many atheist blogs and websites that make fun of the claim about the heart playing a big role in our emotions. They cannot deny the fact that their own research supports it. Because there is not one ingredient in antidepressants that affect the heart muscle in any way. So what is it that causes the heat attack any how? Proof of what is claimed here but denied by those who know such proof proves design and not random chance.

Heart muscle a special design?

The heart has to pump blood 24 hours a day, 7 days a week. Or you will die. In order for the heart to achieve this, it has to have a muscle that never gets tired. If you have ever done anything where you got tired and had to sit and rest, think of how it would be if your heart had to quit beating to take a rest? But yet people can work all day, run marathons, work out, sleep etc… And the heart just keeps on beating. So if the muscle of the heart is not the type that allows the continued use of this organ once life needed it. The species dies and becomes extinct. The heart’s function requires that each ability to be there and fully functioning. And because it will cause death if it is not, there is no room for error that is required for natural selection or survival of the fittest. And “TIME” does not help because “TIME” is not the factor. Being perfect upon it’s creation, the very first time, is. And that leaves no room for it to have evolved. How long can one survive with a heart that does not have this special muscle? One day. Which does not support the millions of years that evolution requires.

Heart rate control disproves evolution?

Upon the heart being able to function comes a problem of how it should function. Such things as:
1) At what rate should it beat?
2) How hard should it beat?
3) What rhythm should it beat?

Why is this important? Continued high blood pressure can damage many things in the body. Every organ can be damaged including the brain. The blood vessels suffer damage as high blood pressure makes them stretch and expand as they try to hold in the high pressure. This damage to the blood vessels causes aneurysms as blood vessels burst. And on the other extreme, to low of a blood pressure can make you pass out, weak, and unable to function. So a variable heart rate controller has to be in place and functioning “correctly” once the heart is functioning. Anyone who has to take meds to control their heart because of a malfunction in this area can relate to what I am speaking of.

Does medical science prove this as well?
Yes, there are several medications available for all kinds of heart problems:
1) Ace inhibitors.
2) Beta blockers.
3) Calcium Channel blockers.
4) Nitrates.
etc…
None of these were available during the evolution process which means the life form would die, or suffer all it’s life

Heart valve design disproves evolution?

Anyone who has had a heart valve replacement can relate to what I am about to speak of. The heart valve has to be designed correctly in order to not allow back flow or leakage as the blood flows in one direction. It also has to not damage the blood cells that flow through it as it opens and closes. If the blood cells become damaged, the body reacts to this as bleeding in that area. At which point blood clots will be made to try and correct what the body thinks is where bleeding is going on. But what this does is send blood clots to the brain, legs, and other organs. Which can cause death or organ damage. This is why heart valve replacement recipients have to take blood thinners to block the clotting process. So what this proves is that if the heart valve is not perfectly designed the very first time, clots will form and the offspring dies. There is no room for “TIME” to be a factor, and therefore natural selection and survival of the fittest will not work when millions of years are not available for something that has to be perfect the very first time.

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Good Fight Ministries
Good Fight Ministries

Joe Schimmel’s Testimony

Blessed Hope Chapel
Wed. May 22, 2019 – Wednesday Night Bible Study led by Pastor Joe Schimmel.
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Ruthann Blizzard Michael Blizzard

Uh, that's not nearly enough manpower for a ship that size.

See ya'll next weekend! Can't wait!

Creation Research Society
Creation Research Society
The Role of Epigenetics in Adaptation, Part 1

The following Matters of Fact column by CRS board member Dr. Jean Lightner appeared in Creation Matters, Vol. 23, No. 3, May/June 2018.

Q.  Does epigenetics play a role in adaptation? 
A.  Physiologist: YES! Evolutionary biologist: Maybe…. 

Adaptation, in the sense that we will discuss, can be defined as changes which help an organism become better suited to its environment. It is related to one of the foundational characteristics of life: the ability to respond to the environment. Physiological adaptation relies on epigenetics, or modifications that can affect gene expression. This does not change the sequence of DNA, but allows genes to be up or down regulated to suit the needs of the organism (see Lightner, 2013). 

There are several known mechanisms of epigenetic regulation (Figure 1): 

1) histone modification (including acetylation, phosphorylation, and methylation) 

2) cytosine methylation in DNA 

3) various non-coding RNA molecules (miRNA, siRNA, piRNA, and lncRNA) 

These mechanisms vary in the timeframe over which they typically act, allowing for both rapid changes and more stable, long-term changes. 

Scientists had assumed that these types of changes could not be inherited by offspring. The basis for this was largely philosophical: the Modern Synthesis (aka Neo-Darwinism) was predicated on the idea that the environment could not direct phenotypic change. Instead, the source of phenotypic variation is claimed to be from random genetic mutations; natural selection then reduces or eliminates less fit variants. To support the conjecture that epigenetic changes are not heritable, some scientists pointed to the observation that DNA methylation patterns are reset in pathways leading to offspring (i.e., germ cell formation and fertilization). However, it is now recognized that the reset of DNA methylation isn’t always complete, and it is not the only mechanism involved in trans-generational epigenetic inheritance (Morgan et al., 1999; Rassoulzadegan et al., 2006). 

For several decades now, it has been known that epigenetic inheritance can provide a source of heritable variation. However, it is not yet clear how often it does so, and what role it plays in adaptation of populations. Research has increased on this important topic, but much remains to be learned. One recent review article identified a web of potential interactions. It also pointed out that understanding patterns of natural epigenetic variation, the causes of that variation, and the consequences of it are necessary to adequately address the role it may have in adaptation (Richards et al., 2017). 

Factors influencing epigenetic variation 

In some studies it appears that DNA methylation differences are associated with underlying genetic differences. This raises the possibility of genetic control of epigenetic variability. It is also possible that a stable epimutation (heritable epigenetic change) could be inherited along with the underlying genetic sequence, thus causing the correlation. It has also been noted that epigenetic changes can influence genetic variation, specifically as it relates to silencing transposable elements, whose movement can change the sequence of a gene or its promoter (Richards et al., 2017). 

Some epimutations appear to arise stochastically. If these are stable over multiple generations, then natural selection may affect the pattern of variation. It is also known that environmental factors can effect heritable epigenetic changes, but the pattern and extent of this is not well known. Significant work needs to be done across different species, especially wild plants and animals, before reasonable generalizations can be made (Balao et al. 2018; Richards et al., 2017). 

FIGURE 1. A chromosome is made up of DNA coiled around proteins, called histones. There are three basic mechanisms by which epigenetic changes can be made. First, the tail of the histone proteins can undergo several types of modification (A), including phosphorylation (Ph), methylation (Me), and acetylation (Ac), that can affect accessibility of specific genes. Secondly, cytosine residues in DNA can be methylated (red dot) or un– methylated (green dot), the details of which are represented in section B of the figure. This affects gene transcription (the copying of DNA to make mRNA). Finally, various microRNAs (C) can bind mRNA to prevent synthesis into proteins. All of these mechanisms play a role in changing gene expression without affecting the DNA sequence. (Illustration is from Gómez-Díaz et al., 2012, and is used herein according to the CC BY license. )

Learn more about creation www.creationresearch.org

The Role of Epigenetics in Adaptation, Part 1

The following Matters of Fact column by CRS board member Dr. Jean Lightner appeared in Creation Matters, Vol. 23, No. 3, May/June 2018.

Q. Does epigenetics play a role in adaptation?
A. Physiologist: YES! Evolutionary biologist: Maybe….

Adaptation, in the sense that we will discuss, can be defined as changes which help an organism become better suited to its environment. It is related to one of the foundational characteristics of life: the ability to respond to the environment. Physiological adaptation relies on epigenetics, or modifications that can affect gene expression. This does not change the sequence of DNA, but allows genes to be up or down regulated to suit the needs of the organism (see Lightner, 2013).

There are several known mechanisms of epigenetic regulation (Figure 1):

1) histone modification (including acetylation, phosphorylation, and methylation)

2) cytosine methylation in DNA

3) various non-coding RNA molecules (miRNA, siRNA, piRNA, and lncRNA)

These mechanisms vary in the timeframe over which they typically act, allowing for both rapid changes and more stable, long-term changes.

Scientists had assumed that these types of changes could not be inherited by offspring. The basis for this was largely philosophical: the Modern Synthesis (aka Neo-Darwinism) was predicated on the idea that the environment could not direct phenotypic change. Instead, the source of phenotypic variation is claimed to be from random genetic mutations; natural selection then reduces or eliminates less fit variants. To support the conjecture that epigenetic changes are not heritable, some scientists pointed to the observation that DNA methylation patterns are reset in pathways leading to offspring (i.e., germ cell formation and fertilization). However, it is now recognized that the reset of DNA methylation isn’t always complete, and it is not the only mechanism involved in trans-generational epigenetic inheritance (Morgan et al., 1999; Rassoulzadegan et al., 2006).

For several decades now, it has been known that epigenetic inheritance can provide a source of heritable variation. However, it is not yet clear how often it does so, and what role it plays in adaptation of populations. Research has increased on this important topic, but much remains to be learned. One recent review article identified a web of potential interactions. It also pointed out that understanding patterns of natural epigenetic variation, the causes of that variation, and the consequences of it are necessary to adequately address the role it may have in adaptation (Richards et al., 2017).

Factors influencing epigenetic variation

In some studies it appears that DNA methylation differences are associated with underlying genetic differences. This raises the possibility of genetic control of epigenetic variability. It is also possible that a stable epimutation (heritable epigenetic change) could be inherited along with the underlying genetic sequence, thus causing the correlation. It has also been noted that epigenetic changes can influence genetic variation, specifically as it relates to silencing transposable elements, whose movement can change the sequence of a gene or its promoter (Richards et al., 2017).

Some epimutations appear to arise stochastically. If these are stable over multiple generations, then natural selection may affect the pattern of variation. It is also known that environmental factors can effect heritable epigenetic changes, but the pattern and extent of this is not well known. Significant work needs to be done across different species, especially wild plants and animals, before reasonable generalizations can be made (Balao et al. 2018; Richards et al., 2017).

FIGURE 1. A chromosome is made up of DNA coiled around proteins, called histones. There are three basic mechanisms by which epigenetic changes can be made. First, the tail of the histone proteins can undergo several types of modification (A), including phosphorylation (Ph), methylation (Me), and acetylation (Ac), that can affect accessibility of specific genes. Secondly, cytosine residues in DNA can be methylated (red dot) or un– methylated (green dot), the details of which are represented in section B of the figure. This affects gene transcription (the copying of DNA to make mRNA). Finally, various microRNAs (C) can bind mRNA to prevent synthesis into proteins. All of these mechanisms play a role in changing gene expression without affecting the DNA sequence. (Illustration is from Gómez-Díaz et al., 2012, and is used herein according to the CC BY license. )

Learn more about creation www.creationresearch.org
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Hidden History of Evolution
Hidden History of Evolution
~ Issac

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Evolution is a lie, based on unsupported assumptions.

Science leads to God
Science leads to God
Had to stop the test, cant have evolution proven wrong. ~ Issac

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180 Movie
180 Movie

Are you one of the over 49,500 views who’s watched “7 Reasons” on YouTube since its release a week ago?

We’ve been so encouraged to read the many online comments, such as this one from YouTube:

"This is THE first time I’ve ever cried (quietly flowing down my cheeks and dripping off my chin kinda tears), regarding the abortion I had 9 years ago. I’ve LITERALLY NEVER felt convicted, not once before…And after watching this.. well, yeh.. I’ve just sat here frowning, now my eyes have dried, and thinking to myself, “Wow, what IS happening to me?!”…I’m a feminist, pro-choice (I thought), equality, love and peace to all kinda person. I guess I better think again................. Mind = blown. Thank you for this video."

If you haven't watched and shared it yet, watch "7 Reasons" free on YouTube at 7ReasonsMovie.com
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Pray for Militant Atheist Page.
Pray for Militant Atheist Page.
n case you did not know this. ~ Issac

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Where is the evolution?
Where is the evolution?
Name: Monito del Monte
Status: Thought to be extinct until its rediscovery.
Information: A remarkable, diminutive marsupial thought to have been extinct until one was discovered in a thicket of Chilean bamboo in the southern Andes.
Thought to exist: 55 million years ago.
Reference: http://historysevidenceofdinosaursandmen.weebly.com/living-fossils.html
The fossilised ankle and ear bones are those of Australias earliest known marsupial, Djarthia, a primitive mouse-like creature that lived 55 million years ago. ..a new study in the journal PLoS ONE [http://www.plosone.org/] has confirmed that Djarthia is also a primitive relative of the small marsupial known as the Monito del Monte - or little mountain monkey - from the dense humid forests of Chile and Argentina.
Reference: http://www.create.unsw.edu.au/news/2008-03-25_monito.html
The monito del monte, Spanish for ‘little bush monkey’, named after its monkey-like partially prehensile tail, is a diminutive marsupial native to South America in the Valdivian temperate rain forests of the southern Andes (Chile and Argentina). It is the only extant species in the ancient order of Microbiotheria. ...Genetic studies show that this species retains the most primitive characteristics of its group, and thus is regarded as a “living fossil.”
reference: http://www.eartharchives.org/articles/scientists-uncover-two-new-species-of-elusive-south-american-marsupial/

Name: Monito del Monte
Status: Thought to be extinct until it's rediscovery.
Information: A remarkable, diminutive marsupial thought to have been extinct until one was discovered in a thicket of Chilean bamboo in the southern Andes.
Thought to exist: 55 million years ago.
Reference: http://historysevidenceofdinosaursandmen.weebly.com/…
"The fossilised ankle and ear bones are those of Australia's earliest known marsupial, Djarthia, a primitive mouse-like creature that lived 55 million years ago. ..a new study in the journal PLoS ONE [http://www.plosone.org/] has confirmed that Djarthia is also a primitive relative of the small marsupial known as the Monito del Monte - or "little mountain monkey" - from the dense humid forests of Chile and Argentina."
Reference: http://create.unsw.edu.au/news/…
"The monito del monte, Spanish for ‘little bush monkey’, named after its monkey-like partially prehensile tail, is a diminutive marsupial native to South America in the Valdivian temperate rain forests of the southern Andes (Chile and Argentina). It is the only extant species in the ancient order of Microbiotheria. ...Genetic studies show that this species retains the most primitive characteristics of its group, and thus is regarded as a “living fossil.”"
reference: http://eartharchives.org/articles/…
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Your picture makes it seem like the two species shown are found 55 Ma apart even though they are both modern species. Rather, it was the genus Djarthia (whose exact taxonomic position is uncertain) that occurs in the Paleocene, as noted in the PLOS paper you provided. This graphic is either a misunderstanding or diliberate misrepresentation of the references cited. May I ask what formal training in paleontology the admin of this page has had?

We didn't claim the skulls were from a 55 million year old fossil, it is the references that claim Monito del Monte is regarded as a living fossil and thought to exist: 55 million years ago.

Colby, please stop spamming the contrasts. There is no need to post the same link multiple times, Thank you.

I was just doing a one shot on each post. I didnt even think anyone even looked at this page anymore. I apologize.

Looks like the Colbinator deleted his post 😭

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